Publications
Browse our publications to find articles, books, reports and papers written by experts in their field.
Many of our researchers make their full text freely available to view and share, supporting research innovation on a global scale.
Recent publications
- Al-Ansari, A. and Robertson, N. P. 2019. Cluster headache: an overview of established and emerging treatments. Journal of Neurology 266 (11), pp.2890-2892. (10.1007/s00415-019-09548-x)
- Barker, R. A. et al., 2019. Designing stem-cell-based dopamine cell replacement trials for Parkinson's disease. Nature Medicine 25 (7), pp.1045-1053. (10.1038/s41591-019-0507-2)
- Bernardi, R. E. et al., 2019. The inhibition of RasGRF2, but not RasGRF1, alters cocaine reward in mice. Journal of Neuroscience 39 (32), pp.6325-6338. (10.1523/JNEUROSCI.1120-18.2019)
- Bhargava, R. , Daughters, K. L. and Rees, D. A. 2019. Oxytocin therapy in hypopituitarism: challenges and opportunities. Clinical Endocrinology 90 (2), pp.257-264. (10.1111/cen.13909)
- Bourbon Teles, J. et al., 2019. Myelin breakdown in human Huntington's disease: multi-modal evidence from diffusion MRI and quantitative magnetization transfer. Neuroscience 403 , pp.79-92. (10.1016/j.neuroscience.2017.05.042)
- Cavenagh, A. , Chatterjee, S. and Davies, W. 2019. Behavioural and psychiatric phenotypes in female carriers of genetic mutations associated with X-linked ichthyosis. PLoS ONE 14 (2) e0212330. (10.1371/journal.pone.0212330)
- Crawford, K. et al. 2019. Medical consequences of pathogenic CNVs in adults: Analysis of the UK Biobank. Journal of Medical Genetics 56 , pp.131-138. (10.1136/jmedgenet-2018-105477)
- Davies, W. 2019. An analysis of Cellular Communication Network (CCN) proteins as candidate mediators of postpartum psychosis risk. Frontiers in Psychiatry 10 876. (10.3389/fpsyt.2019.00876)
- Davies, W. and Wilkinson, L. 2019. Editorial overview: special issue on epigenetics and genomic imprinting. Current Opinion in Behavioral Sciences 25 , pp.iii-v. (10.1016/j.cobeha.2018.12.006)
- Drakesmith, M. et al. 2019. Genetic risk for schizophrenia and developmental delay is associated with shape and microstructure of midline white-matter structures. Translational Psychiatry 9 (1) 102. (10.1038/s41398-019-0440-7)
- Drew, C. J. G. et al. 2019. A protocol for a randomised controlled, double-blind feasibility trial investigating fluoxetine treatment in improving memory and learning impairments in patients with mesial temporal lobe epilepsy: Fluoxetine, Learning and Memory in Epilepsy (FLAME trial). Pilot and Feasibility Studies 5 (1) 87. (10.1186/s40814-019-0474-x)
- Egmond, M. E. et al., 2019. Variable interpretation of the dystonia consensus classification items compromises its solidity. Movement Disorders 34 (3), pp.317-320. (10.1002/mds.27627)
- Fjodorova, M. et al. 2019. CTIP2-regulated reduction in PKA-dependent DARPP32 phosphorylation in human medium spiny neurons: implications for Huntington’s disease. Stem Cell Reports 13 (3-6), pp.448-457. (10.1016/j.stemcr.2019.07.015)
- Föcking, M. et al., 2019. Complement pathway changes at age 12 are associated with psychotic experiences at age 18 in a longitudinal population-based study: evidence for a role of stress. Molecular Psychiatry (10.1038/s41380-018-0306-z)
- Fung, W. K. W. and Peall, K. J. 2019. What is the role of the cerebellum in the pathophysiology of dystonia?. Journal of Neurology 266 (6), pp.1549 -1551. (10.1007/s00415-019-09344-7)
- Huckins, L. M. et al., 2019. Gene expression imputation across multiple brain regions provides insights into schizophrenia risk. Nature Genetics 51 , pp.659-674. (10.1038/s41588-019-0364-4)
- Humby, T. and Davies, W. 2019. Brain gene expression in a novel mouse model of postpartum mood disorder. Translational Neuroscience 10 (1), pp.168-174. (10.1515/tnsci-2019-0030)
- Kedia, G. et al., 2019. So Pretty! The neural correlates of self-other vs familiar-other attractiveness comparisons. Social Neuroscience 14 (1), pp.41-52. (10.1080/17470919.2017.1397544)
- Kopczynska, M. et al. 2019. Complement system biomarkers in first episode psychosis. Schizophrenia Research 204 , pp.16-22. (10.1016/j.schres.2017.12.012)
- Lancaster, T. M. et al. 2019. Structural and functional neuroimaging of polygenic risk for schizophrenia: a recall-by-genotype-based approach. Schizophrenia Bulletin 45 (2), pp.405-414. (10.1093/schbul/sby037)
- Lansdown, A. et al. 2019. Regional cerebral activation accompanies sympathoexcitation in women with polycystic ovary syndrome. Journal of Clinical Endocrinology and Metabolism 104 (9), pp.3614-3623. (10.1210/jc.2019-00065)
- Lipp, I. et al. 2019. Comparing MRI metrics to quantify white matter microstructural damage in multiple sclerosis. Human Brain Mapping 40 (10), pp.2917-2932. (10.1002/hbm.24568)
- Manivannan, S. et al. 2019. The effectiveness of virtual reality interventions for improvement of neurocognitive performance post-traumatic brain injury: a systematic review. Journal of Head Trauma Rehabilitation 34 (2), pp.E52-E65. (10.1097/HTR.0000000000000412)
- Nadadhur, A. G. et al., 2019. Neuron-glia interactions increase neuronal phenotypes in tuberous sclerosis complex patient iPSC-derived models. Stem Cell Reports 12 (1)(10.1016/j.stemcr.2018.11.019)
- Niarchou, M. et al. 2019. Psychiatric disorders in children with 16p11.2 deletion and duplication. Translational Psychiatry (10.1038/s41398-018-0339-8)
- Noakes, Z. et al. 2019. Human pluripotent stem cell-derived striatal interneurons: differentiation and maturation in vitro and in the rat brain. Stem Cell Reports 12 (2), pp.191-200. (10.1016/j.stemcr.2018.12.014)
- Plumbly, W. et al. 2019. L-type voltage-gated calcium channel regulation of in vitro human cortical neuronal networks. Scientific Reports 9 13810. (10.1038/s41598-019-50226-9)
- Ranson, A. et al. 2019. Top-down suppression of sensory cortex in an NMDAR hypofunction model of psychosis. Schizophrenia Bulletin 45 (6), pp.1349-1357. (10.1093/schbul/sby190)
- Richards, A. et al. 2019. Associations between schizophrenia genetic risk, anxiety disorders and manic/hypomanic episode in a New Zealand longitudinal population cohort study. British Journal of Psychiatry 214 (2), pp.96-102. (10.1192/bjp.2018.227)
- Salunkhe, G. et al., 2019. The impact of the COMT genotype and cognitive demands on facets of intra-subject variability. Brain and Cognition 132 , pp.72-79. (10.1016/j.bandc.2019.03.002)
- Savory, K. and Syed, Y. A. 2019. Advances in the understanding of cellular pathogenesis associated with Autism Spectrum Disorder. Journal for ReAttach Therapy and Developmental Diversities (10.26407/2019jrtdd.1.21)
- Schwarz, E. et al., 2019. Reproducible grey matter patterns index a multivariate, global alteration of brain structure in schizophrenia and bipolar disorder. Translational Psychiatry 9 (1), pp.-. 12. (10.1038/s41398-018-0225-4)
- Silva, A. I. et al. 2019. Reciprocal white matter changes associated with copy number variation at 15q11.2 BP1-BP2: A diffusion tensor imaging study. Biological Psychiatry 85 (7), pp.-. (10.1016/j.biopsych.2018.11.004)
- Srinivasan, R. et al. 2019. Mental health and behavioural problems in children with XXYY: a comparison with intellectual disabilities. Journal of Intellectual Disability Research 63 (5), pp.477-488. (10.1111/jir.12607)
- Sykes, L. et al. 2019. Genetic variation in the psychiatric risk gene CACNA1C modulates reversal learning across species. Schizophrenia Bulletin 45 (5), pp.1024-1032. (10.1093/schbul/sby146)
- Toste, C. et al. 2019. No effect of genome-wide significant schizophrenia risk variation at the DRD2 locus on the allelic expression of DRD2 in post-mortem striatum. Molecular Neuropsychiatry 5 (4), pp.212-217. (10.1159/000501022)
- Vasalauskaite, A. , Morgan, J. and Sengpiel, F. 2019. Plasticity in adult mouse visual cortex following optic nerve injury. Cerebral Cortex 29 (4), pp.1767-1777. (10.1093/cercor/bhy347)
- Vorobyov, V. et al., 2019. Loss of midbrain dopamine neurons and altered apomorphine EEG effects in the 5xFAD mouse model of Alzheimer's disease. Journal of Alzheimer's Disease 70 (1), pp.241-256. (10.3233/JAD-181246)
- Whale, R. et al., 2019. Factor analyses differentiate clinical phenotypes of idiopathic and interferon-alpha-induced depression.. Brain, Behavior, and Immunity 80 , pp.519-524. (10.1016/j.bbi.2019.04.035)
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