Dr Carmen Van Den Berg

2019

• Lau, Y. H. L., Gliddon, C. and Van Den Berg, C. 2019. Development of a competitive ELISA and genotype technique to determine caffeine pharmacokinetics and CYP1A2 status in humans. The British Student Doctor Journal 3(3), pp. 38-40. (10.18573/bsdj.117)

2018

• Delafontaine, M.et al. 2018. Venom from Bothrops lanceolatus, a snake species native to Martinique, potently activates the complement system. Journal of Immunology Research 2018, article number: 3462136. (10.1155/2018/3462136)
• Manzoni-de-Almeida, D.et al. 2018. Loxosceles venom Sphingomyelinase D activates human blood leukocytes: Role of the complement system. Molecular Immunology 94, pp. 45-53. (10.1016/j.molimm.2017.12.009)

2017

• Stott, M.et al. 2017. Post-translational modification of complement anaphylatoxins during chronic neutrophilic inflammation. Molecular Immunology 89, pp. 167., article number: 103. (10.1016/j.molimm.2017.06.141)
• Okamoto, C.et al. 2017. Tetracycline Reduces Kidney Damage Induced by Loxosceles Spider Venom. Toxins 9(3), article number: 90. (10.3390/toxins9030090)

2016

• Myamoto, D. T.et al. 2016. Characterization of the gene encoding component C3 of the complement system from the spider Loxosceles laeta venom glands: phylogenetic implications. Immunobiology 221(9), pp. 953-963. (10.1016/j.imbio.2016.05.009)
• Cristina de Oliveira-Lima, K.et al. 2016. Microcirculation abnormalities provoked by Loxosceles spiders' envenomation. Toxicon 116, pp. 35-42. (10.1016/j.toxicon.2015.08.005)
• Manzoni de Almeida, D.et al. 2016. Loxosceles Sphingomyelinase D induces leukocyte activation which is partially complement dependent: Similarities to endotoxic shock. Toxicon 116, pp. 77-77. (10.1016/j.toxicon.2016.01.019)
• Correa, M. A.et al. 2016. Sphingomyelinase D from loxosceles laeta venom induces the expression of MMP7 in human keratinocytes: contribution to dermonecrosis. PLoS ONE 11(4), article number: e0153090. (10.1371/journal.pone.0153090)
• Ho, P. L.et al. 2016. Characterization of a gene coding for the complement system component FB from loxosceles laeta spider venom glands. PLoS ONE 11(1), article number: e0146992. (10.1371/journal.pone.0146992)

2015

• Giles, J. L.et al. 2015. Response to comment on 'Functional analysis of a complement polymorphism (rs17611) associated with rheumatoid arthritis'. Journal of Immunology 195(1), pp. 4-4. (10.4049/jimmunol.1500968)
• Stover, C. M.et al. 2015. A serine protease isolated from the bristles of the Amazonic caterpillar, Premolis semirufa, is a potent complement system activator. PLoS ONE 10(3), article number: e0118615. (10.1371/journal.pone.0118615)
• Giles, J.et al. 2015. Functional analysis of a complement polymorphism (rs17611) associated with rheumatoid arthritis. The Journal of Immunology 194(7), pp. 3029-3034. (10.4049/jimmunol.1402956)

2014

• Giles, J. L.et al. 2014. Functional analysis of a complement polymorphism (rs17611) associated with Rheumatoid Arthritis. Immunology 143(s2), pp. 181-182. (10.1111/imm.12407)
• Tambourgi, D. V. and van den Berg, C. W. 2014. Animal venoms/toxins and the complement system. Molecular Immunology 61(2), pp. 153-162. (10.1016/j.molimm.2014.06.020)
• Van Den Berg, C.et al. 2014. Mechanism of neutrophil dysfunction: neutrophil serine proteases cleave and inactivate the C5a receptor. The Journal of Immunology 192(4), pp. 1787-1795. (10.4049/jimmunol.1301920)

2013

• Pidde-Queiroz, G.et al. 2013. P-I snake venom metalloproteinase is able to activate the complement system by direct cleavage of central components of the cascade. PLoS Neglected Tropical Diseases 7(10), pp. e2519. (10.1371/journal.pntd.0002519)

2012

• Van Den Berg, C. W.et al. 2012. C5a receptor is cleaved by metalloproteases induced by sphingomyelinase D from Loxosceles spider venom. Immunobiology 217(9), pp. 935-941. (10.1016/j.imbio.2012.01.005)
• Palmer, E.et al. 2012. Roles of promoter and 3′ untranslated motifs in expression of the human C5a receptor. Molecular Immunology 52(2), pp. 88-95. (10.1016/j.molimm.2012.04.012)
• Harris, R., Van Den Berg, C. W. and Bowen, D. J. 2012. ASGR1 and ASGR2, the genes that encode the asialoglycoprotein receptor (Ashwell receptor), are expressed in peripheral blood monocytes and show interindividual differences in transcript profile. Molecular Biology International 2012 (10.1155/2012/283974)
• Villas-Boas, I. M.et al. 2012. Premolis semirufa (Walker, 1856) Envenomation, disease affecting rubber tappers of the amazon: searching for caterpillar-bristles toxic components. Plos Neglected Tropical Diseases 6(2), article number: e1531. (10.1371/journal.pntd.0001531)
• Tanaka, G. D.et al. 2012. Micrurus snake venoms activate human complement system and generate anaphylatoxins. BMC Immunology 13, pp. 1-7., article number: 4. (10.1186/1471-2172-13-4)

2010

• Tambourgi, D. V., Gonçalves-de-Andrade, R. M. and Van Den Berg, C. W. 2010. Loxoscelism: From basic research to the proposal of new therapies. Toxicon 56(7), pp. 1113-1119. (10.1016/j.toxicon.2010.01.021)
• Gray, L. C., Hughes, T. R. and Van Den Berg, C. W. 2010. Binding of human antigen R (HuR) to an AU-rich element (ARE) in the 3' untranslated region (3' UTR) reduces the expression of decay accelerating factor (DAF). Molecular Immunology 47(16), pp. 2545-2551. (10.1016/j.molimm.2010.07.002)
• Pidde-Queiroz, G.et al. 2010. Human complement activation and anaphylatoxins generation induced by snake venom toxins from Bothrops genus. Molecular Immunology 47(16), pp. 2537-2544. (10.1016/j.molimm.2010.07.003)

2009

• de Santi Ferrara, G. I.et al. 2009. SMase II, a new sphingomyelinase D from Loxosceles laeta venom gland: Molecular cloning, expression, function and structural analysis. Toxicon 53(7-8), pp. 743-753. (10.1016/j.toxicon.2009.02.013)

2008

• Hakobyan, S.et al. 2008. Complement factor H binds to denatured rather than to native pentameric C-reactive protein. Journal of Biological Chemistry 283(45), pp. 30451-30460. (10.1074/jbc.M803648200)
• Donev, R. M.et al. 2008. Modulation of CD59 expression by restrictive silencer factor-derived peptides in cancer immunotherapy for neuroblastoma [RETRACTED]. Cancer Research 68(14), pp. 5979-5987. (10.1158/0008-5472.CAN-07-6828)
• de Almeida, D. M.et al. 2008. A new anti-loxoscelic serum produced against recombinant sphingomyelinase D: results of preclinical trials. American Journal of Tropical Medicine and Hygiene 79(3), pp. 463-470.

2007

• Van Den Berg, C. W.et al. 2007. Loxosceles spider venom induces the release of thrombomodulin and Endothelial Protein C Receptor: implications for the pathogenesis of intravascular coagulation as observed in loxoscelism. J Thromb Haemost 5, pp. 989-995. (10.1111/j.1538-7836.2007.02382.x)

2005

• Taylor, K. E., Giddings, J. C. and Van Den Berg, C. W. 2005. C-reactive protein-induced in vitro endothelial cell activation is an artefact caused by azide and lipopolysaccharide. Arteriosclerosis Thrombosis and Vascular Biology 25(6), pp. 1225-1230. (10.1161/01.ATV.0000164623.41250.28)
• Van Den Berg, C. W. and Capey, S. 2005. Porcine complement regulators protect aortic smooth muscle cells poorly against human complement-induced lysis and proliferation: consequences for xenotransplantation. Xenotransplantation 12(3), pp. 217-226. (10.1111/j.1399-3089.2005.00217.x)

2004

• Van Den Berg, C. W., Taylor, K. E. and Lang, D. 2004. C-reactive protein-induced in vitro vasorelaxation is an artefact caused by the presence of sodium azide in commercial preparations. Arteriosclerosis Thrombosis and Vascular Biology 24(10), pp. e168-171. (10.1161/01.ATV.0000142807.92781.d9)

2002

• Hiscox, S. E.et al. 2002. GPI-anchored GFP signals Ca2+ but is homogeneously distributed on the cell surface. Biochemical and Biophysical Research Communications 293(2), pp. 714-721. (10.1016/S0006-291X(02)00280-2)

2001

• Hiscox, S. E. and Van Den Berg, C. W. 2001. Characterisation of cell activation through cyt2 and cyt1 tail of MCP. Molecular Immunology 38(2-3), pp. 96-97.

1995

• Van Den Berg, C. W.et al. 1995. Exogenous CD59 incorporated into U937 cells through its glycosyl phosphatidylinositol anchor becomes associated with signalling molecules in a time dependent manner. Biochemical Society Transactions 23(2), pp. 269S. (10.1042/bst023269s)