Ewch i’r prif gynnwys

Chwilio rhaglenni ymchwil

Filter results

1-26 o 26 canlyniadau chwilio

Enw Cymhwyster Dull Type

Biomeddygaeth

Mae Biofeddygaeth yn faes ymchwil lle galwch ganolbwyntio eich astudiaethau fel rhan o’n cyfres o raglenni ymchwil Biowyddorau.

PhD, MPhil, MD Amser llawn, rhan-amser Maes

Niwrowyddoniaeth

Mae’r adran Niwrowyddoniaeth yn mynd ar drywydd amrywiaeth eang o ymchwil niwrofiolegol, yn cwmpasu lefelau moleciwlaidd i ymddygiadol.

PhD, MPhil, MD Amser llawn, rhan-amser Maes

Meddygaeth

The School of Medicine offers research degrees in the medical disciplines such as cancer, immunology, infection, immunity, neurosciences, mental health and population medicine.

PhD, MPhil, MD Amser llawn, rhan-amser Rhaglen

Biowyddorau Moleciwlaidd

Mae Biowyddorau Moleciwl yn canolbwyntio ar fecanweithiau moleciwlaidd sy’n sail i swyddogaeth biolegol.

PhD, MPhil, MD Amser llawn, rhan-amser Maes

Bioffiseg Adeileddol

Mae Bioffiseg Adeileddol yn faes ymchwil y gallwch chi ganolbwyntio eich astudiaethau yn ein rhaglen PhD Gwyddorau’r Golwg ynddo.

PhD Amser llawn, rhan-amser Maes

Canser a Geneteg

Bydd y rhaglen hon yn arwain at radd ymchwil mewn canser a/neu geneteg y gellid eu defnyddio mewn amrywiaeth o leoliadau academaidd, clinigol a diwydiannol.

PhD, MPhil, MD Amser llawn, rhan-amser Maes

Haint ac Imiwnedd

I gynnig gwybodaeth eang ac arbenigedd ym mhob agwedd ar brosesau clefyd imiwnolegol yn seiliedig ar y lefel cellog a moleciwlaidd, gyda chryfderau mewn imiwnedd cynhenid, imiwnoleg canser, bioleg celloedd-T a heintiau bacteriol a firaol.

PhD, MPhil, MD Amser llawn, rhan-amser Maes

Gwyddorau Biofeddygol a'r Geg

Mae ein Gwyddorau Biofeddygol a’r Geg yn faes ymchwil lle galwch ganolbwyntio eich astudiaethau fel rhan o’n cyfres o raglenni ymchwil Deintyddiaeth (MPhil, PhD).

PhD, MPhil Amser llawn, rhan-amser Maes

Ymchwilio Clinigol a Gwyddorau'r Golwg

Mae Ymchwilio Clinigol a Gwyddorau’r Golwg yn faes ymchwil y gallwch chi ganolbwyntio eich astudiaethau yn ein rhaglen PhD Gwyddorau’r Golwg ynddo.

PhD Amser llawn, rhan-amser Maes

Astudio clefyd niwroseiciatrig dynol mewn niwronau dynol diffygiol DLG2

Mae'r prosiect PhD hwn mewn Meddygaeth yn ceisio deall rôl y DLG2 yn ystod datblygiad niwral a niwronau aeddfed gan ddefnyddio amrywiaeth o dechnegau.

Prosiect

Integration of genetic and functional data to identify drug targets and enhance risk prediction

Integrating brain expression and protein-protein interaction data with genomic data identified a network of immune-related genes implicated in Alzheimer’s disease (AD) susceptibility.

Prosiect

Characterising DNA damage repair in Huntington’s disease

Parkinson’s disease (PD) is the second most common neurodegenerative disorder and results from the selective loss of dopaminergic neurons within the Substantia nigra pars compacta.

Prosiect

Detecting dysfunction in the single-cell gene networks underlying Parkinson’s disease

Parkinson’s disease (PD) is the second most common neurodegenerative disorder and results from the selective loss of dopaminergic neurons within the Substantia nigra pars compacta.

Prosiect

Microglial NFAT signalling and Alzheimer’s disease genetic risk networks

In this project, you will develop skills in molecular biological, cell culture, genome biology and bioinformatics.

Prosiect

The molecular basis of cognitive flexibility

This project examines transcriptomes in key brain regions at critical periods to determine the molecular bases of flexible behaviour.

Prosiect

Investigating peroxisome dysregulation in Alzheimer’s disease

In this project you will first determine how peroxisomes respond to the specific pathogenic proteins associated with AD in vivo.

Prosiect

Machine Learning for Prediction in Alzheimer's Disease: Identifying Novel Biologically Valid Diagnostic Categories to Inform Precision Medicine

The project will involve investigating the ability of machine learning.

Prosiect

Targeting LAG3+ T cells for Cancer Immunotherapy

This study will look at the control of adaptive immunity in health and inflammation by human blood and mucosal gamma/delta T cells.

Prosiect

Alzheimer’s disease: Understanding the interaction of microglia risk genes

The aim of this project is to understand the interdependence of specific risk alleles and highlight the possibility of adjunctive therapies targeting different ‘arms’ of the immune cells to treat the disease.

Prosiect

Investigating the childhood neurodevelopmental origins of adult mental illness

This PhD utilises new data generated by a Wellcome Trust Collaborative Award between Cardiff and Bristol Universities to test hypotheses on why child NDDs show links with later adult mental illness.

Prosiect

Flexible vectors for immunotherapy against cancer and pathogens

This project aims to determine pathways that can drive the growth of particular types of NK and T cells that are optimised to provide better protection from disease. We will use cytomegalovirus and leukaemic cells as systems of analysis.

Prosiect

Role of a mouse ortholog of CR1 (crry) in the 3xTG model of Alzheimer’s Disease (AD)

In order to explore further roles of Crry, we have generated and established a 3xTG/Crry-/- colony, but have not yet had resources to explore further the impact of Crry deficiency in this model.

Prosiect

Enhancing immune stimulation for novel anti-cancer viral vaccine vectors

We will combine cutting-edge proteomics with molecular virology and immunology to determine how HCMV induces such strong responses, enabling us to generate optimised vaccine vectors.

Prosiect

Optimizing adult mental health outcomes in children with neurodevelopmental problems: interplay of social and genetic factors

This PhD project provides a unique opportunity to develop advanced methodological and analytic skills in the field of developmental psychopathology.

Prosiect

School experiences in children at high genetic risk of mental health problems

This project will provide important insights into why children with a genomic disorder struggle in school.

Prosiect

Molecular mechanisms of CRISPR Cas9 nickase-induced CAG/CTG repeat contraction: Implications for gene editing in expanded repeat disorders

The aim of this project is to understand how contractions occur at the molecular level such that we can make a potential gene editing-based treatment safer and more efficient.

Prosiect